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 Post subject: Re: VIX Trading
PostPosted: 9/21/18 11:06 
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Joined: 1/28/12 01:21
Posts: 65544

我找几个人打听了,好像都不看好。说要去几毛钱。你怕不怕?

silenceman2013 wrote:
下周是见证奇迹的时刻了。
dave_liu wrote:

你的AMRN还拿着?都要9月底了,还没考试。

silenceman2013 wrote:
当然。而且是超级大仓位。否则打算骗你什么。 可惜主席不出现了


 
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 Post subject: Re: VIX Trading
PostPosted: 9/21/18 12:21 
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因为那些人都没吃过 VASCEPA, 总把它当作一般的鱼油
dave_liu wrote:

我找几个人打听了,好像都不看好。说要去几毛钱。你怕不怕?



 
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 Post subject: Re: VIX Trading
PostPosted: 9/21/18 12:23 
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另外估计他们都不知道 10年前还有个 Jelis Trial (Japan)


 
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 Post subject: Re: VIX Trading
PostPosted: 9/21/18 12:33 
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另外VASCEPA 能成功地原因不是它能 升高好胆固醇。而是它能修复免疫系统。降低衰老带来的机能下降。


 
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 Post subject: Re: VIX Trading
PostPosted: 9/21/18 13:20 
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Why R-I will be an enormous success...

The answer is very simple...All the other cardio drugs simply treat the manifestations and the symptoms of the disease...this includes every drug from insulin to statins...None of them treat the root case of the disease. This is analogous to having a leak in one of your car's tires...you can correct the problem temporarily by simply pumping the tire back up..but to fix the problem you need to repair the leak.

EPA is the only drug in the cardo field that fixes the leak which is elevated systemic inflammation...Attacking the lipid parameters is just blowing the tire back up...

":>) JL


 
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 Post subject: Re: VIX Trading
PostPosted: 9/21/18 14:15 
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见证科学的力量时刻了。这实验都搞了快7年。

For those of you here who know literally NOTHING about Vascepa (EPA), if you understand these two paragraphs from Dr. Sears, you know enough:

"The ultimate goal of using omega-3 fatty acids is the reduction of cellular inflammation. Since eicosanoids derived from arachidonic acid (AA), an omega-6 fatty acid, are the primary mediators of cellular inflammation, EPA becomes the most important of the omega-3 fatty acids to reduce cellular inflammation for a number of reasons. First, EPA is an inhibitor of the enzyme delta-5-desaturase (D5D) that produces AA (1). The more EPA you have in the diet, the less AA you produce. This essentially chokes off the supply of AA necessary for the production of pro-inflammatory eicosanoids (prostaglandins, thromboxanes, leukotrienes, etc.). DHA is not an inhibitor of this enzyme because it can’t fit into the active catalytic site of the enzyme due to its larger spatial size. As an additional insurance policy, EPA also competes with AA for the enzyme phospholipase A2 necessary to release AA from the membrane phospholipids (where it is stored). Inhibition of this enzyme is the mechanism of action used by corticosteroids. If you have adequate levels of EPA to compete with AA (i.e. a low AA/EPA ratio), you can realize many of the benefits of corticosteroids but without their side effects. That’s because if you don’t release AA from the cell membrane then you can’t make inflammatory eicosanoids. Because of its increased spatial dimensions, DHA is not a good competitor of phospholipase A2 relative to EPA. On the other hand, EPA and AA are very similar spatially so they are in constant competition for the phospholipase A2 enzyme just as both fatty acids are in constant competition for the delta-5 desaturase enzyme. This is why measuring the AA/EPA ratio is such a powerful predictor of the state of cellular inflammation in your body.

The various enzymes (COX and LOX) that make inflammatory eicosanoids can accommodate both AA and EPA, but again due to the greater spatial size of DHA, these enzymes will have difficulty in converting DHA into eicosanoids. This makes DHA a poor substrate for these key inflammatory enzymes. Thus DHA again has little effect on cellular inflammation whereas EPA can have a powerful impact."


 
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 Post subject: Re: VIX Trading
PostPosted: 9/21/18 15:03 
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Joined: 12/16/08 18:39
Posts: 5840
我想买点自己吃,怎么买?http://www.trader1688.com/bb/images/smilies/icon_biggrin.gif
silenceman2013 wrote:
见证科学的力量时刻了。这实验都搞了快7年。 For those of you here who know literally NOTHING about Vascepa (EPA), if you understand these two paragraphs from Dr. Sears, you know enough: "The ultimate goal of using omega-3 fatty acids is the reduction of cellular inflammation. Since eicosanoids derived from arachidonic acid (AA), an omega-6 fatty acid, are the primary mediators of cellular inflammation, EPA becomes the most important of the omega-3 fatty acids to reduce cellular inflammation for a number of reasons. First, EPA is an inhibitor of the enzyme delta-5-desaturase (D5D) that produces AA (1). The more EPA you have in the diet, the less AA you produce. This essentially chokes off the supply of AA necessary for the production of pro-inflammatory eicosanoids (prostaglandins, thromboxanes, leukotrienes, etc.). DHA is not an inhibitor of this enzyme because it can’t fit into the active catalytic site of the enzyme due to its larger spatial size. As an additional insurance policy, EPA also competes with AA for the enzyme phospholipase A2 necessary to release AA from the membrane phospholipids (where it is stored). Inhibition of this enzyme is the mechanism of action used by corticosteroids. If you have adequate levels of EPA to compete with AA (i.e. a low AA/EPA ratio), you can realize many of the benefits of corticosteroids but without their side effects. That’s because if you don’t release AA from the cell membrane then you can’t make inflammatory eicosanoids. Because of its increased spatial dimensions, DHA is not a good competitor of phospholipase A2 relative to EPA. On the other hand, EPA and AA are very similar spatially so they are in constant competition for the phospholipase A2 enzyme just as both fatty acids are in constant competition for the delta-5 desaturase enzyme. This is why measuring the AA/EPA ratio is such a powerful predictor of the state of cellular inflammation in your body. The various enzymes (COX and LOX) that make inflammatory eicosanoids can accommodate both AA and EPA, but again due to the greater spatial size of DHA, these enzymes will have difficulty in converting DHA into eicosanoids. This makes DHA a poor substrate for these key inflammatory enzymes. Thus DHA again has little effect on cellular inflammation whereas EPA can have a powerful impact."


 
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 Post subject: Re: VIX Trading
PostPosted: 9/21/18 15:25 
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OmegaVia EPA 500, 纯度差些。效果似乎也有。就是现在断货了。
cxyz wrote:
我想买点自己吃,怎么买?http://www.trader1688.com/bb/images/smilies/icon_biggrin.gif


 
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 Post subject: Re: VIX Trading
PostPosted: 9/21/18 18:20 
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Is OmegaVia a AMRN's brand? Doesn't seem it. Good review though.
silenceman2013 wrote:
OmegaVia EPA 500, 纯度差些。效果似乎也有。就是现在断货了。
cxyz wrote:
我想买点自己吃,怎么买?http://www.trader1688.com/bb/images/smilies/icon_biggrin.gif


 
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 Post subject: Re: VIX Trading
PostPosted: 9/22/18 21:21 
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Joined: 10/18/15 23:19
Posts: 91
AHA18
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 Post subject: Re: VIX Trading
PostPosted: 9/24/18 05:39 
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恭喜小板栗。发大了。

silenceman2013 wrote:
当然。而且是超级大仓位。否则打算骗你什么。 可惜主席不出现了
last wrote:
R u on this med?
silenceman2013 wrote:
9月出结果


 
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 Post subject: Re: VIX Trading
PostPosted: 9/24/18 06:27 
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恭喜板栗吃个胖子!!
dave_liu wrote:

恭喜小板栗。发大了。

silenceman2013 wrote:
当然。而且是超级大仓位。否则打算骗你什么。 可惜主席不出现了
last wrote:
R u on this med?


 
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 Post subject: Re: VIX Trading
PostPosted: 9/24/18 07:47 
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恭喜大家 。不过这个还能涨。
dave_liu wrote:

恭喜小板栗。发大了。

silenceman2013 wrote:
当然。而且是超级大仓位。否则打算骗你什么。 可惜主席不出现了
last wrote:
R u on this med?


 
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 Post subject: Re: VIX Trading
PostPosted: 9/24/18 08:18 
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Posts: 65544

目标多少?
silenceman2013 wrote:
恭喜大家 。不过这个还能涨。
dave_liu wrote:

恭喜小板栗。发大了。

silenceman2013 wrote:
当然。而且是超级大仓位。否则打算骗你什么。 可惜主席不出现了


 
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 Post subject: Re: VIX Trading
PostPosted: 9/24/18 08:35 
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Joined: 8/3/10 21:33
Posts: 9703
I have to say. It is an important moment for science. I recently resumed my investing activities. run one after another, VKTX, ACAD, AMRN ....


eat more purified fish oil and fish - such as live fish oil as atlantic mackeral.


Congrats.


 
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